Transgenderism in fish is caused by common human drugs and why should we care
Transgenderism or gender dysphoria is easily becoming a growing phenomenon is society, but what is causing children as young as two to say that they in the wrong body? The newly discovered transgender phenomenon in fish populations could explain what is could be happening to this generation. Here we discuss why we should care about recent reports that transgenderism in fish is caused by common human drugs.
This might be a delicate topic to talk about, but this is the moment to address transgenderism, since scientific research has linked the use of common human drugs, such as contraceptive pills to transgenderism in fish. Professor Charles Tyler, of the University of Exeter, was able to observe in his study that “…oestrogens found in some plastics affect the valves in the heart.”. Also, tests showed 20 per cent of male freshwater fish, such as roach, at 50 sites had feminine characteristics.”
So, the question that begs to be asked is: If transgenderism in fish is caused by common human drugs, what is it doing to our body and more importantly to the hormonal and genetic development of upcoming generations?
Here are some scientific studies about the connection between hormones, neurological development, gender and sexual behaviour
If you look into older reports about gender bending chemicals like estrogens, you will see that scientists have been warning that manmade pollutants, which have escaped into the environment, mimic the female sex hormone oestrogen. The males of species including fish, amphibians, birds, and reptiles have been feminised by exposure to sex hormone disrupting chemicals and have been found to be abnormally making egg yolk protein.
Interestingly, a study on the effects of estrogen on male mice, has found that while the reproductive system of male mice homozygous for a mutation in the estrogen receptor (ER) gene (ER knock-out; ERKO) appears normal at the anatomical level. These males are infertile, indicating an essential role for ER-mediated processes in the regulation of male reproduction. In addition to that, other studies have found a relationship between prenatal endocrine influences of gonadal steroids like testosterone, on sexual orientation and on sexually differentiated childhood behavior. For example: the impact of exposure to the hormone at the appropriate time can persist across the lifespan. These early, time-linked, persistent effects are thought to occur because hormones direct some aspects of neural development during early life, influencing cell survival, neuroanatomical connectivity and neurochemical specification.
While researches on human patients, have linked prenatal exposure to androgens to transgendersm in male to female transgenders. Other studies have observed that developing female rats with estradiol, or other estrogens, does not have feminizing effects, but the opposite. The effects of estrogens resemble those of testosterone, increasing male-typical sexual behavior and the volume of the SDN-POA, and reducing female-typical sexual behavior. Additional evidence that estrogen plays a role in neurobehavioral masculinisation early in life comes from evidence that blocking conversion of androgen to estrogen, or blocking estrogen receptors, has some of the same effects as castration, even in the presence of high levels of testosterone.
The effects of chemical pollutants in society?