It may be possible to delay aging
In mice and roundworm adding a coenzyme can extend life and postpone the onset of aging processes and this is anticipated to also be true in people.
People have always dreamed of being able to stop the aging process and live indefinitely. New research shows that it may be possible to actually delay the aging process. The University of Copenhagen reports a substance which has the potential to postpone the aging process has been found. This substance is called coenzyme NAD+.
Coenzyme NAD+ may help postpone aging
Coenzyme NAD+ plays a primary role in the process of aging. It has been observed by researchers that adding this substance to mice and roundworm can extend life while also postponing the beginning of processes of aging. Research which has been pursued at the Center for Healthy Aging and the American National Institute of Health shows that in the future this new knowledge will be able to be used to help patients suffering from Alzheimer’s and Parkinson’s disease.
Researchers worldwide have been attempting to better understand aging processes. As people live longer and longer the quality of life with aging has become a bigger issue. This new understanding may help to postpone the physical aging process and extend life. Professor Vilhelm Bohr has said it was surprising to observe that the addition of NAD+ delayed the aging processes of the cells and prolonged life in a mouse model and in worms.
Coenzyme NAD+ repairs to the DNA and poorly functioning mitochondria
It has been observed by the researchers that coenzyme NAD+ repairs to the DNA and poorly functioning mitochondria. This is significant because it has been determined a primary process in aging is the capacity of our cells to keep our DNA intact. The power stations of cells, the mitochondria, also have been affect on aging processes.
Professor Bohr says it has been shown in this study that there are age-dependent decreases in the level of NAD+. This decrease is far greater for organisms which display early aging and which lack DNA repairs. He goes on to explain that previous studies have shown us that a decrease in the level of NAD+ results in errors of metabolism, neurodegeneration and aging.
This study has been published in the journal Cell Metabolism. Although the effect of NAD+ has just been examined on model organisms and not yet administered to patients, the researchers anticipate seeing the same effect in people. This is because cell repair processes are universal for the cells of all living organisms. It is therefore actually possible this new knowledge will be able to help delay physical aging processes and prevent illnesses such as Parkinson’s disease and Alzheimer’s.